Australian Labradoodles UK
Australian Labradoodle Health Testing
We are a small Australian Labradoodle breeder with strong ethics and a big heart. Breeding healthy good, quality family dogs.
All our breeding Australian Labradoodles are fully health tested. We have all their health test certificates to show you. We would welcome the opportunity to discuss all the health tests with you. We urge you to always ask your chosen breeder for test results and always ask to see the test certificates for both parents of the puppies. The results of these tests are really important. A puppy being health checked by a vet is not enough by any means. It is imperative that the Australian Labradoodle parents are clear of genetic diseases and have undergone hip, elbow and eye evaluations.
Hip scored by the BVA or OFA
Elbow scored by the BVA.
They will undergo physical Eye testing tests for the following inherited eye diseases:
MRD – Multifocal Retinal Dysplasia
TRD – Total Retinal Dysplasia
GPRA – Generalised Progressive Retinal Atrophy
CPRA – Central Progressive Retinal Atrophy
DNA Tests - VWD (Von Willebrand )
DNA Tests - PRA (Progressive Retinal Atrophy)
DNA Test - DM (Degenerative Myelopathy)
DNA Test - IC (Improper Coat)
DNA Test - EIC (Exercise-induced Collapse)
All of our Australian Labradoodle puppies are from health tested parents. Every Australian Labradoodle puppy is health checked by our vet, microchipped, wormed and vaccinated, All our puppies come with a puppy pack full of information and advice.
We send all our DNA testing to America to the world-renowned Paw Print Genetics
Paw Print Genetics offers the largest selection of tests for inherited diseases.
Breeding dogs should always be tested before it is bred so that the breeder is aware of any potential genetic disorders that it could pass to its offspring.
Testing is an ongoing part of our breeding program, All of our dogs are clear of genetic diseases.
Paw Print have the Highest Industry Standards and Accuracy
Paw Print Say:-
Our laboratory is staffed with expertly trained geneticists, veterinarians, and technicians. We are equipped with the latest testing technology and analyze each mutation with two independent methods to provide you the highest accuracy in the industry.
All mutations offered are based on the published, medical literature
Board-certified geneticist by the American Board of Medical Genetics on staff
Each mutation is tested twice, with two independent methods
All results are reviewed and reported by both a PhD geneticist and a veterinarian
Majority of test results accepted by the Orthopedic Foundation for Animals
Diagnostic-grade DNA extracted from a variety of accepted sample types
We hope that the disease information below helps you to see why it is so essential that your Australian Labradoodle puppy comes from health tested parents.
Degenerative Myelopathy (DM)
Affected dogs usually present in adulthood with gradual muscle Atrophy and loss of coordination, typically beginning in the hind limbs due to degeneration of the nerves. The condition is not typically painful for the dog but will progress until the dog is no longer able to walk. The gait of dogs affected with degenerative myelopathy can be difficult to distinguish from the gait of dogs with hip dysplasia, arthritis of other joints of the hind limbs, or intervertebral disc disease. Late in the progression of the disease, dogs may lose faecal and urinary continence, and the forelimbs may be affected. Affected dogs may fully lose the ability to walk 6 months to 2 years after the onset of symptoms.
Exercise-Induced Collapse (EIC)
It is an inherited neuromuscular disorder affecting Australian Labradoodles. EIC presents as exercise intolerance in apparently healthy dogs. Affected dogs are usually diagnosed before two years of age and appear normal during low to moderately strenuous activity. However, shortly after 5-20 minutes of strenuous exercise, affected dogs will begin to walk with a wobbly, uncoordinated gait that often only affects the hind limbs. Dogs remain mentally alert and are not in pain during episodes of EIC. In some circumstances, the symptoms of EIC can progress to full body weakness with low muscle tone (flaccid paralysis), confusion, loss of consciousness, seizures and, very rarely, death. The episodes typically last 5-10 minutes, and most dogs will completely recover within 15-30 minutes.
Progressive retinal Atrophy, cone-Rod dystrophy 4 (PRA-crd4)
is an inherited eye disease affecting dogs. PRA-crd4 occurs as a result of degeneration of both rod and cone type Photoreceptor Cells of the Retina, which are important for vision in dim and bright light, respectively. Affected dogs can show symptoms of vision loss or have signs of retinal disease on a veterinary ophthalmologic exam by 3 years of age. However, age of onset varies significantly in PRA-crd4 affected dogs and has been reported from 1 to 15 years of age.
Progressive retinal Atrophy, progressive Rod-cone degeneration (PRA-prcd)
is a late-onset, inherited eye disease affecting dogs. PRA-prcd occurs as a result of degeneration of both rod and cone type Photoreceptor Cells of the Retina, which are important for vision in dim and bright light, respectively. Evidence of retinal disease in affected dogs can first be seen on an Electroretinogram around 1.5 years of age for most breeds, but most affected dogs will not show signs of vision loss until 3 to 5 years of age or later. The rod type cells are affected first, and affected dogs will initially have vision deficits in dim light (night blindness) and loss of peripheral vision. Over time affected dogs continue to lose night vision and begin to show visual deficits in bright light. Other signs of progressive retinal atrophy involve changes in reflectivity and the appearance of a structure behind the retina called the Tapetum that can be observed on a veterinary eye exam.
Von Willebrand disease (VWD) is an inherited bleeding disorder affecting Australian Labradoodles. Dogs affected with VWDI have less than half of the normal level of von Willebrand coagulation factor (vWf), which is an essential protein needed for normal blood clotting. There is variability in the amount of vWf made such that not all dogs with two copies of the Mutation are equally affected. Dogs that have less than 35% of the normal amount of vWf generally have mild to moderate signs of a bleeding disorder. Affected dogs may bruise easily, have frequent nosebleeds, bleed from the mouth when juvenile teeth are lost, and experience prolonged bleeding after surgery, trauma, or estrus. Dogs may show signs of lameness or stiffness if bleeding occurs in the joints or muscles. Less often, the bleeding may be severe enough to cause death. Due to the variable severity of the disorder, affected dogs may not be identified until surgery is performed, or trauma occurs, at which time excessive bleeding is noted. Veterinarians performing surgery on known affected dogs should have ready access to blood banked for transfusions.
Improper coat (IC) Locus determines if a dog will have longer hair around the muzzle and eyebrows (facial furnishings) or a lack of furnishings (called improper coat in breeds for which facial furnishings are standard). A DNA variant in the RSPO2 gene, which functions in hair follicle development, results in the production of furnishings in multiple breeds and is found in all wire-haired dogs. A third variant at the IC locus has been identified and associated with “weak” furnishings that develop gradually into sparse and much less pronounced furnishings. A puppy born from parents whom both carry IC may not look like an Australian Labradoodle. They may present as having a hair coat like a labrador or lab cross the coat of these pups will shed.
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